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Work in the Lamitina Lab utilizes the model organism C. elegans to define mechanisms of age-related neurodegenerative diseases, such as ALS. The lab also investigates how stress responses are coordinated at the organismal level. The accumulation of conformationally aberrant proteins is a focus of both research projects. Through our work, we hope to understand if 'natural' mechanisms for opposing protein misfolding (i.e. stress responses) might be leveraged for the treatment of these currently incurable neurological diseases.